Difference between revisions of "Young RE, et al, Journal (2004), cited as Ref 54 in DOI: 10.1016/j.clim.2020.108427 (Q11297)"

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(‎Created claim: Eighth Author string (P156): Nourshargh S, #quickstatements; #temporary_batch_1593104117513)
(‎Created claim: DOI Identifier (P138): 10.4049/jimmunol.172.7.4493, #quickstatements; #temporary_batch_1593106448087)
 
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Neutrophil elastase (NE)-deficient mice demonstrate a nonredundant role for NE in neutrophil migration generation of proinflammatory mediators and phagocytosis in response to zymosan particles in vivo (English)
Property / title: Neutrophil elastase (NE)-deficient mice demonstrate a nonredundant role for NE in neutrophil migration generation of proinflammatory mediators and phagocytosis in response to zymosan particles in vivo (English) / rank
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172
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Property / DOI Identifier: 10.4049/jimmunol.172.7.4493 / rank
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Latest revision as of 17:34, 25 June 2020

Cited in: "COVID-19 pathophysiology: A review" Clin Immunol; 2020 Apr 20 108427.
Language Label Description Also known as
English
Young RE, et al, Journal (2004), cited as Ref 54 in DOI: 10.1016/j.clim.2020.108427
Cited in: "COVID-19 pathophysiology: A review" Clin Immunol; 2020 Apr 20 108427.

    Statements

    Journal
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    2004
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    Young RE
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    Thompson RD
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    Larbi KY
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    La M
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    Roberts CE
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    Shapiro SD
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    Perretti M
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    Nourshargh S
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    Neutrophil elastase (NE)-deficient mice demonstrate a nonredundant role for NE in neutrophil migration generation of proinflammatory mediators and phagocytosis in response to zymosan particles in vivo (English)
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    172
    0 references